Lancet Study Shows Boosters Increase Chance of Getting Covid

And Natural Immunity is Superior

A recent study published in The Lancet looked at “Population immunity of natural infection, primary-series vaccination, and booster vaccination in Qatar during the COVID-19 pandemic”

The study was conducted on the population of Qatar between 1 July 2020 and 30 November 2022 and looked at national databases for testing, vaccination, hospitalisation and death. This included a total of 899,441 cases and 10,709,791 controls for previous infections, 796,255 cases and 9,126,914 controls for primary vaccination and 568,661 cases and 5,342,301 controls for boosters.

Both natural and vaccine immunity waned over time but waning was particularly rapid for vaccine population immunity.

What was most concerning was that booster vaccine effectiveness went into negative values, suggesting immune imprinting. This has been seen in different data sets over and over again with vaccinated individuals MORE likely to catch Covid due to their vaccines.

As you can see in the chart above, booster effectiveness was virtually non-existent by February 2022 and into negative territory by April 2022. It has stayed negative ever since then.

We have known about this for years now but this study confirms everything we have seen before. Covid vaccines quickly go into negative efficacy and in doing so make the vaccinated more susceptible to getting infected.

Of course, the authors had to conclude that this means more boosters should be given to the vulnerable. But this is to be expected and such wording has to be included in studies to get published nowadays. I’m surprised it didn’t start with ‘vaccines are safe and effective and have saved millions of lives’ because that is the usual line.

Some good news from the study is again information that we knew right from the beginning of the pandemic. Similar to common-cold coronaviruses, infection probably results in life-long immunity against severe reinfection. “ Long-term immune protection against severe COVID-19 could contribute to a benign pattern of infection that is perhaps not dissimilar to that of common-cold coronaviruses”.